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<article article-type="research-article" dtd-version="1.3" xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xml:lang="ru"><front><journal-meta><journal-id journal-id-type="publisher-id">ometendo</journal-id><journal-title-group><journal-title xml:lang="ru">Ожирение и метаболизм</journal-title><trans-title-group xml:lang="en"><trans-title>Obesity and metabolism</trans-title></trans-title-group></journal-title-group><issn pub-type="ppub">2071-8713</issn><issn pub-type="epub">2306-5524</issn><publisher><publisher-name>Endocrinology Research Centre</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="doi">10.14341/omet2013437-42</article-id><article-id custom-type="elpub" pub-id-type="custom">ometendo-6389</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="ru"><subject>Статьи</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="en"><subject>Articles</subject></subj-group></article-categories><title-group><article-title>Вторичное ожирение</article-title><trans-title-group xml:lang="en"><trans-title>Secondary obesity. Part 2</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Babarina</surname><given-names>M B</given-names></name></name-alternatives><bio xml:lang="ru"><p>к.м.н., ст.н.с. отделения терапии эндокринопатий лечебно-диагностического отдела</p></bio><email xlink:type="simple">endocrin@mail.ru</email></contrib><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Fadeeva</surname><given-names>M I</given-names></name></name-alternatives><bio xml:lang="ru"><p>н.с. отделения терапии эндокринопатий лечебно-диагностического отдела</p></bio><email xlink:type="simple">fm2004@bk.ru</email></contrib><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Savelieva</surname><given-names>L V</given-names></name></name-alternatives><bio xml:lang="ru"><p>к.м.н., зав. отделением терапии эндокринопатий лечебно-диагностического отдела</p></bio><email xlink:type="simple">larisa.savelieva@inbox.ru</email></contrib></contrib-group><pub-date pub-type="collection"><year>2013</year></pub-date><pub-date pub-type="epub"><day>15</day><month>12</month><year>2013</year></pub-date><volume>10</volume><issue>4</issue><issue-title>№4 (2013)</issue-title><fpage>37</fpage><lpage>42</lpage><permissions><copyright-statement>Copyright &amp;#x00A9; Babarina M.B., Fadeeva M.I., Savelieva L.V., 2013</copyright-statement><copyright-year>2013</copyright-year><copyright-holder xml:lang="ru">Babarina M.B., Fadeeva M.I., Savelieva L.V.</copyright-holder><copyright-holder xml:lang="en">Babarina M.B., Fadeeva M.I., Savelieva L.V.</copyright-holder><license xml:lang="ru" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>Данная работа распространяется под лицензией Creative Commons Attribution 4.0.</license-p></license><license xml:lang="en" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>This work is licensed under a Creative Commons Attribution 4.0 License.</license-p></license></permissions><self-uri xlink:href="https://www.omet-endojournals.ru/jour/article/view/6389">https://www.omet-endojournals.ru/jour/article/view/6389</self-uri><abstract><p>Глюкокортикоиды, пролактин и соматотропный гормон играют важную роль в регуляции метаболического гомеостаза. Гиперкортицизм, гиперпролактинемия и дефицит гормона роста приводят к увеличению массы тела за счет висцерального жира с преимущественным его перераспределением по абдоминальному типу. Механизмы развития ожирения при различных эндокринопатиях неодинаковы. Адекватная коррекция гормональных нарушений, как правило, сопровождается снижением массы тела и улучшением метаболических показателей.</p></abstract><trans-abstract xml:lang="en"><p>Glucocorticoids, prolactin and growth hormone play an important role in the regulation of metabolic homeostasis. Hypercortisolism, hyperprolactinemia and growth hormone deficiency lead to an increase in body weight due to visceral fat accumulation. Mechanisms of development of obesity in various endocrine diseases are different. Adequate correction of hormonal disorders usually is accompanied by a decrease in body weight and the improvement of metabolic parameters.</p></trans-abstract><kwd-group xml:lang="ru"><kwd>ожирение</kwd><kwd>кортизол</kwd><kwd>пролактин</kwd><kwd>соматотропный гормон</kwd></kwd-group><kwd-group xml:lang="en"><kwd>obesity</kwd><kwd>cortisol</kwd><kwd>prolactin</kwd><kwd>growth hormone</kwd></kwd-group></article-meta></front><back><ref-list><title>References</title><ref id="cit1"><label>1</label><citation-alternatives><mixed-citation xml:lang="ru">Нейроэндокринология. Клинические очерки. Под редакцией Е.И. Маровой. Ярославль: «ДИА-пресс», 1999.</mixed-citation><mixed-citation xml:lang="en">Нейроэндокринология. Клинические очерки. Под редакцией Е.И. Маровой. Ярославль: «ДИА-пресс», 1999.</mixed-citation></citation-alternatives></ref><ref id="cit2"><label>2</label><citation-alternatives><mixed-citation xml:lang="ru">Ожирение: этиология, патогенез, клинические аспекты. 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